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Biology of Cancer - Cancer Currents Blog

Cancer biology research news, with context from experts at NCI and elsewhere. Topics include cancer metastasis, the tumor microenvironment, and new targets for cancer therapies.

  • Icon of a colon with three tumors on the right side. A message bubble with three dots points from the colon tumors. On the other side, an icon of a yellowish liver with a message bubble coming from it.
    • By Nadia Jaber

    When colorectal cancer spreads to the liver, it can be very difficult to treat. Cancer is more likely to invade the liver when patients have fatty liver disease. A recent study places the blame on “message bubbles” called extracellular vesicles that are released by the liver.

  • An illustration of a tumor in the pancreas
    • By Carmen Phillips

    A new study finds that pancreatic cancer cells have a ready way to overcome a lack of glucose, a frequent occurrence in this disease. They use another fuel source: a molecule called uridine. Findings from a related study suggest other cancers do as well.

  • An illustration of a synapse between two neurons
    • By Sharon Reynolds

    Researchers have found that the aggressive brain cancer glioblastoma can co-opt the formation of new synapses to fuel its own growth. This neural redirection also appears to play a role in the devastating cognitive decline seen in many people with glioblastoma.

  • A scanning electron micrograph showing chromosomes and ecDNA
    • By Carmen Phillips

    A new study shows for the first time that a circular form of DNA, called ecDNA, is present in precancerous tissue and not just cancer cells. The study also suggested that when ecDNA is present in Barrett’s esophagus, that tissue is almost certain to become cancer.

  • An illustration depicting chromothripsis and its outcome, including ecDNA formation
    • By Edward Winstead

    Researchers have identified a mechanism by which cancer cells develop specific genetic changes needed to become resistant to targeted therapies. They also showed that this process, called non-homologous end-joining (NHEJ), can potentially be disrupted.

  • Illustration of ETV6 and EWS-FLI1 binding to DNA
    • By Linda Wang

    The protein ETV6 appears to promote tumor growth by affecting the behavior of the EWS-FLI1 fusion protein that drives most Ewing sarcomas. The research groups that made the discovery hope it leads to a targeted therapy for the aggressive childhood cancer.

  • Two microscopic images are shown, each displaying complex cellular structures and tissues. The top image features a network of cells and fibers mostly in blue, green, and purple, with some areas highlighted in pink. The bottom image also shows a dense arrangement of cells and fibers with similar staining, but presents a different pattern and distribution of the colored elements.
    • By Elia Ben-Ari

    An experimental drug, MRTX1133, shrank tumors or halted their growth in several mouse models of human pancreatic cancer with KRAS G12D mutations, a new study shows. The models included one that is genetically engineered to closely mimic the human disease.

  • An illustration of T cells attacking inflamed heart tissue by binding to alpha-myosin.
    • By Carmen Phillips

    In people with cancer treated with immune checkpoint inhibitors, a rare, but often fatal, side effect is inflammation in the heart, called myocarditis. Researchers have now identified a potential chief cause of this problem: T cells attacking a protein in heart cells called α-myosin.

  • An anatomic illustration of a Roux-en-Y gastric bypass
    • By Sharon Reynolds

    A new study suggests that people with obesity who had bariatric surgery had a much lower risk of five common cancers that aren’t related to hormone levels, including lung, colorectal, and esophageal cancer.

  • Illustration of T cells attacking a cancer cell.
    • By Carmen Phillips

    In a new study, researchers showed that cancer cells with mutations in the IDH1 gene release large amounts of a metabolite called D-2HG. Once outside of the cells, D-2HG acts like force field by neutralizing nearby immune cells.

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